神道 Shéndào | Voie divine [merveilleux]) (Nguyen Van Nghi 1971) Voie du Shen (Pan 1993, Ellis 1989, Laurent 2000) Voie de l'esprit (Lade 1994) | Spiritual Passage (Li Ding 1992) Spirit Path (Ellis 1989) |
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Chōng dào | 衝道 (1) | Xun Jing (Guillaume 1995) voie des battements (cardiaques)(Laurent 2000) |
Zàng shū | 脏腧 (臟腧) (2) | Qian Jin Yao Fang (Guillaume 1995) point des Zang (Laurent 2000) |
Zhuàng shū | 状输 [狀輸] (3) | selon le Ji sheng, ce serait une erreur de transcription (Guillaume 1995) |
Items de localisation
Ligne médiane postérieure
D5-D6
Xinshu
Acupuncture | Moxibustion | Source |
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Ce point est interdit à la puncture | Faire 3 moxas | Yi xue ru men (Guillaume 1995) |
Il est interdit de puncturer ce point | Da cheng (Guillaume 1995) | |
Selon Tong ren, appliquer de 7x 7 à 100 cônes de moxa, au maximum. Selon Ming xia, appliquer 3 cônes de moxa, puncturer à 0,5 distance. Selon Qian jin yao fang, appliquer 5 cônes de moxa | Zhen jiu ju ying (Guillaume 1995) | |
Piquer obliquement vers le haut, à 0,5-1 distance | Cautériser 3-7 fois, chauffer 5-10 minutes | Roustan 1979 |
Puncture inclinée entre 0,5 et 1 distance de profondeur | Cautérisation avec 3 à 5 cônes de moxa, moxibustion pendant 5 à 15 minutes. | Guillaume 1995 |
Piqûre oblique vers le haut de 0,5 à 1 cun | Moxas : 3 ; chauffer 10 à 20 mn | Laurent 2000 |
Sensation de puncture
Sécurité
Classe d'usage | - | point secondaire |
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Indication | Association | Source |
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Dans la Malaria qui débute par la nuque et le dos | 16VG + 11VG + 11V | Su wen (Guillaume 1995) |
Chaleur du corps, céphalée, fièvre intermittente | 11VG + 4VC | Qian jin (Guillaume 1995) |
Accès de frissons et de fièvre | 11VG + 3C | Zi sheng jing (Guillaume 1995). |
Épilepsie de type vent-feng xian | 11VG + 15V | Bai zheng fu (Guillaume 1995) |
Perte de mémoire | 11VG + 21Rn + 7P + 43V | Zi sheng jing (Guillaume 1995) |
Épilepsie de type vent-feng xian | 11VG + 15V | Zhen jiu ju ying-Bai zheng fu (Guillaume 1995) |
Objective To investigate the efficacy of transverse insertion of thick needles into the Du meridian in treating rat ischemic facial paralysis and preliminarily explore the possible mechanism by which it produces a therapeutic effect. Methods Sixty Wistar rats were randomly allocated to groups A (transverse insertion of thick needles), B (basic Western drugs), C (blank control) and D (sham operation), 15 rats each. A rat model of ischemic facial paralysis was made using a modified vascular occlusion method in all the rats. Facial nerve deficits were scored in every group of rats during treatment. At 1 day after model making, group A was treated by transverse insertion into point Shendao (11VG) and 4-hour retention of needle, once daily, for a total of 14 days; group B was treated by an oral gavage of prednisone and intraperitoneal injection of vitamin B12, once daily, for a total of 14 days. Groups C and D were not treated. In 4 rats randomly chosen at 3, 7 or 14 days after model making, facial nerve concomitant intrinsic vascular tissue HIF-1αprotein was measured by an immunohistochemical method; serum NO, by enzyme-linked immunosorbent assay; serum ET content, by radioimmunoassay. Results After 7 days of treatment, the neurological deficit score increased gradually in groups A and B and there was a statistically significant difference compared with after model making in the two groups (P<0. 01). After 7 days of treatment, there was a statistically significant difference in the neurological deficit score in groups A and B compared with groups C and D (P<0. 05) and between groups C and D (P<0. 05). After 14 days of treatment, there was a statistically significant difference in the neurological deficit score between group A or B and group C (P<0. 05) and between group B or C and group D (P<0. 05). In group A, there was a statistically significant difference in the neurological deficit score after 14 days of treatment compared with after 7 days of treatment (P<0. 05). After 3, 7 and 14 days of treatment, there was a statistically significant difference in HIF-1αpMOD value in groups A, B and C compared with group D (P 0. 01). After 3 and 7 days of treatment, there was a statistically significant difference in HIF-1αpMOD value between group A or B and group C (P<0. 05). In groups A and B, there was a statistically significant difference in HIF-1αpMOD value after 3 and 14 days of treatment compared with after 7 days of treatment (P<0. 05). After 3 and 7 days of treatment, there was a statistically significant difference in serum NO content between group A or B and group D (P<0. 01). In groups A and B, there was a statistically significant difference in serum NO content after 7 days of treatment compared with after 3 days of treatment (P<0.05). After 3 and 7 days of treatment, there was a statistically significant difference in serum ET content in groups A, B and C compared with group D (P< 0. 05). In groups A and B, there was a statistically significant difference m serum ET content after 7 days of treatment compared with after 3 days of treatment (P<0. 05). Conclusions Transverse insertion of thick needles into the Du meridian can effectively promote rat's recovery from ischemic facial paralysis. The mechanism by which it produces a therapeutic effect may be regulating the expression of tissue HIF-1αpa and the NO and ET contents of serum.